Download e-book for kindle: Active Defense Mechanisms in Plants by R. Heitefuss (auth.), R. K. S. Wood (eds.)

By R. Heitefuss (auth.), R. K. S. Wood (eds.)

ISBN-10: 1461583098

ISBN-13: 9781461583097

ISBN-10: 146158311X

ISBN-13: 9781461583110

A NATO complex research Institute on "Active Defence Mechanisms in crops" used to be held at Cape Sounion, Greece, 21 April - three might 1981. It succeeded the same Institute held at Porte Conte, Sardinia in 1975 on "Specificity in Plant ailments. " What are lively defence mechanisms within the context of plant disorder during which a plant, the host, can be broken by way of a pathogen? Defence mechanisms contain homes of the host that reduce this harm. The mechanisms are passive once they are self sufficient of the pathogen. they're energetic after they stick to adjustments within the host attributable to the pathogen. hence for a fungal pathogen, telephone partitions of a better plant that are lignified ahead of an infection will be a passive defence mechanism in the event that they diminished harm by way of impeding development of the fungus. mobile partitions identified to turn into lignified as a reaction to the pathogen will be an energetic defence mechanism if it have been demonstrated that this reaction reduced harm. The papers and discussions at this complex learn Institute have been approximately energetic defence mechanisms in larger vegetation, in general econo­ mically very important crop crops, opposed to fungi, micro organism and viruses as pathogens. Taking the microorganisms first it's a truism yet one who bears repeating that even if vegetation normally develop in shut organization with a wide selection of fungi and micro organism, usually of sorts that may be pathogens, they hardly develop into diseased, at the least now not sufficiently so that it will allure notice.

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O Incubation at 25°C (h) Figure I. Temporal relationships between necrosis, accumulation of phytoalexins and inhibition of fungal growth Hypocotyls of P. vuZgaris, cv. Kievitsboon Koekoek were inoculated with C. Zindemuthianum, incubated at 160 C for o 72 hours and transferred to 25 C. Infected cell death, phytoalexin concentration and length of intracellular hyphae were measured. BAILEY Another critical approach necessary in order that firm conclusions may be drawn is to compare the physiological and biochemical effects of phytoalexins on fungal hyphae in vit~o with the state of fungus in resistant plant cells.

J. (1977). Membrane mobility and the concanavalin A binding system of the plasmalemma of higher plant protoplasts. PZanta 136, 253 - 259. 5. M. J. (1978). Ultrastructural cell wall alterations in immune cotton leaves inoculated with Xanthomonas maZvaaearum. PhytopathoZogy 68, 1015 1021. 6. A. A. (1978). A cytochemical study of lipolytic activity in Bremia Zaatuaae Regel during germination of the conidium and penetration of the host. PhysioZogiaaZ PZant PathoZogy 12, 289 - 296. 7. , RALPH, B. R.

1959). Hypersensitivity. In: P~t Pathology Vol. 1, Ed. G. E. Dimond, pp. 469 - 519. Academic Press, New York. 35. OGLE, H. F. (1971). Quantitative studies of the post-penetration phase of infection by Puccinia gpaminis tPitici. Annals of Applied Biology 67, 309 - 319. 36. , SEXTON, R. W. (1978). Ultrastructural observations on the development of the hypersensitive reaction in leaves of Phaseolus vulgapis cv. Red Mexican inoculated with Pseudomonas phaseolicola (race 1). Physiological Plant Pathology 12, 151 - 157.

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Active Defense Mechanisms in Plants by R. Heitefuss (auth.), R. K. S. Wood (eds.)

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